IVIg-mediated amelioration of murine ITP via FcgammaRIIb is not necessarily independent of SHIP-1 and SHP-1 activity.

1. Karahalios DG, Rekate HL, Khayata MH, Apostolides PJ. Elevated intracranial venous pressure as a universal mechanism in pseudotumour cerebri of varying etiologies. Neurology. 1996;46:198-202. 2. Johnston I, Kollar C, Dunkley S, Assaad N, Parker G. Cranial venous outflow obstruction in the pseudotumour cerebri syndrome: incidence, nature and relevance. J Clin Neurosci. 2002;9:273-278. 3. Sussman J, Leach M, Greaves M, Malia R, Davies-Jones GAB. Potentially prothrombotic abnormalities of coagulation in benign intracranial hypertension. J Neurol Neurosurg Psychiatry. 1997;62:229-233. 4. Martinelli I, Battaglioli T, Pedotti P, Cattaneo M, Mannucci PM. Hyperhomocysteinemia in cerebral venous thrombosis. Blood. 2003;102:1363-1366. 5. Ludemann P, Nabavi DG, Junker R, et al. Factor V Leiden mutation is a risk factor for cerebral venous thrombosis. Stroke. 1998;29:2507-2510. 6. Martinelli I, Sacchi E, Landi G, Taioli E, Duca F, Mannucci PM. High risk of cerebral vein thrombosis in carriers of a prothrombin gene mutation and in users of oral contraceptives. N Engl J Med. 1998;338:1793-1797. 7. Van de Meer FJ, Koster T, Vandenbroucke JP, Briet E, Rosendaal FR. The Leiden thrombophilia study. Thromb Haemost. 1997;78:631-635. 8. Ginsberg JS, Wells PS, Brill-Edwards P, et al. Antiphospholipid antibodies and venous thromboembolism. Blood. 1995;86:3685-3691.